Preliminary scientific tests indicate conolidine might inhibit unique ion channels, minimizing neuronal excitability and limiting pain alerts. This mechanism is especially relevant in neuropathic pain, exactly where abnormal signaling leads to persistent pain. Also, conolidine seems to influence G protein-coupled receptor (GPCR) pathways integral to soreness notion. Computational docking https://johnr047lmk9.blogoxo.com/profile